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Splash of plasma had welded their base hinges. woetjans must have expected that, because at least six mauls slammed rhythmically into the masts within seconds of the jam. both began to lift. s3 continued normally, but the pump pressure driving s4 flatlined when the antenna had only elevated a few degrees. a hydraulic line--scored by plasma, fractured by an injudicious hammerblow, or simply filled with the cussed determination of machines to fail--had broken. brady, brady, brady, don't you know you done wrong? the mast resumed its rise, again within seconds of the initial failure. the bosun must already have rigged tackle to blocks at the head of adjacent, previously extended, masts. daniel felt a rush of affection. by god! he wasn't going to let woetjans throw her life away. not even if saving her required a sincerely offered threat to blow her head off if she didn't obey. antenna starboard 4 locked into place and, without further hesitation, unfurled its suit of sails. the princess cecile was wearing nearly eighty percent of her rig, an unusual event made more remarkable by the battle damage that alone prevented the figure being even higher. atoms stripped of electrons and accelerated by repulsion up the bore of a plasma cannon had velocities little short of light speed, but negligible mass. their ravening touch would destroy the first layer of any matter they collided with, but they wouldn't penetrate. damage beyond the target's outer layer was a result of transmitted impact--which in the case of sail fabric was almost zero. after the battleship's initial volley had removed the sails, further bolts would scour the hull. at point-blank range, fluxes intended to change the course of missiles approaching at .6 c would make short work of a corvette. you bust into my bar when the game was on the astrogation computer changed the sails' potentials as programmed; daniel checked the results against the plan and his instinct. all was well. he grinned. if that was the phrase to use under the circumstances. "three minutes to reentry to normal space!" dorst said. the riggers, their job completed, were clanging back within the sissie's hull. the inner airlock opened outside the bridge. one figure stepped through, lt. mon lifting off the helmet of his rigging suit. he closed the hatch behind him. you sprung my latch and you broke my door . catching daniel's eye, mon shouted, "hogg's staying in the lock with woetjans. says it's as good a place as the next, he figures." daniel thought of his short, dumpy servant and the rangy bosun. under the circumstances the two were an ideal pair: they understood one another perfectly. missed communications had killed more people than ever malice dreamed of doing. "daniel?" adele said. she'd waited until she saw his attention drawn away from the calculations on his display. "when we return to normal space, i intend to direct the other ships, the escort ships? direct them to return to tanais in the name of admiral chastelaine. i doubt they'll obey, but i thought it might confuse them. is that all right?" daniel opened a window in his holographic display so that he could meet adele's eyes without a fog of light between them. she looked worried, concerned about having overstepped her proper authority.
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I want a 10% price rollback. now." seller: "i hear what you are saying.but what are we really talking about? is this a recognition thing for you? an empowerment thing? do you feel my company has not treated you with enough respect in the past? let's be honest with each other." buyer: "thanks, pal, but if i'd wanted psychotherapy, i woulda' called a shrink. i'm here to talk pricing. and i'm in a hurry. what's it going to be?" mr. thomas's book is thus primarily about tactics in the core "science" process of negotiation: getting the other side to make concessions in a zero-sum game. he is somewhat skeptical about the "creative" side: it is important, he admits, but the returns should not be exaggerated. while professor shell's book suffers from excessive complexity -- it is chock-full of frameworks and structures, with four steps, four types of negotiation, five styles, six foundations, and no single organizing principle -- mr. thomas's book is refreshingly straightforward. he offers seven critical rules, four important but obvious rules, 10 "nice to do" rules, and a bit about ethics and special situations. to its credit, the book always explains the logic of its advice. it is not especially original, but it is punchy, no-nonsense, and humane. although mr. thomas has a more competitive style than professor shell, he places just as much emphasis on the importance on relationships and is actually tougher on ethics, stating categorically that negotiators should never lie. as for wisdom, he admonishes the reader not to "hose" the other side -- not to use every bit of leverage and every tactic in the book to get the best possible deal. given widely prevalent norms of fair play, such "win-lose" deals really pay off only when there will be no subsequent relationship. instead, the thomas-guided negotiator seeks a win-win deal, meaning that both parties feel they have achieved their objectives even if they have not maximized their gain. ironically, this discussion of purpose is the one place where the hard-edged and ruthless mr. thomas turns out to be a bit too benevolent. major retail chains, for. The caribbean stud online poker games rule progressive jackpot side bet average return jackpot meter the caribbean stud poker house edge will depend directly on the progressive meter at any given moment and free online video poker. Let's say you set your hand the following way: this is an error because the top hand isn't at least jacks and twos.
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I felt like i was in a coffin." these are more than theoretical concerns. on april 8, 2000, 19 marines were killed in a training exercise when a v-22 descended too fast and crashed near tucson. it was the third v-22 to crash -- seven people were killed in two previous crashes. in december 2000, four more marines, including the program's most experienced pilot, were killed in a crash caused by a burst hydraulic line and software problems. these accidents led to program delays to make design changes. but as tests resumed, so have the problems. three engine fires occurred recently because of problems related to hydraulic lines. in march 2006, a computer problem led an idling v-22 to suddenly take off on its own. it then slammed into the ground, breaking off its right wing. all 54 v22's were grounded for weeks in february because of a faulty computer chip. in preparation for deployment, the pentagon ran tests last year in the new mexico desert, similar to the climate of iraq. in january, the pentagon wrote about frequent failures with various parts and systems. the reason: "extended exposure to the desert operating environment." because of these problems, mr. coyle, the former pentagon weapons tester, predicted the marines will use the v-22 to ferry troops from one relatively safe spot to another, like a flying truck. "they don't want to have a `black hawk down, ' " he said. "that would kill the program. of course, it was not designed to be a truck. it was designed to be used in combat. Ome women diagnosed with early breast who also received herceptin, 87.1% were. cancer remain at higher risk of the difference in disease-free survival in the recurrence because of unique characteristics hera study 2 years after diagnosis was 8% of their cancer. one of these high-risk higher 77.4% to 85.8% ; among women who features is referred to as "gene amplification" had received herceptin in addition to the the production of multiple copies of a gene ; . other therapies. the b-31 n9831 results when a gene such as the her-2 gene is also showed that the addition of herceptin amplified, the amount of protein made by reduced the risk of death by 33%.1 the the gene is greatly increased; this is referred results from bcirg 006 will be presented to as "over-expression." about 25% of breast at the san antonio breast cancer cancers amplify the her-2 gene, which symposium in december 2005, but makes the cancer more aggressive and puts preliminary results indicate that there was women diagnosed with it "her-2 positive benefit from adding herceptin to breast cancer" ; at increased risk for chemotherapy in this trial as well. recurrence, even with standard herceptin administered with chemotherapy and hormonal therapy chemotherapy does carry some risk for treatments. charles e. geyer, jr., m.d. serious cardiac toxicity. in the b-31, n9831, researchers at genentech developed a and bcirg 006 studies, 2-4% of patients national surgical adjuvant monoclonal antibody, a biologic therapy, who received herceptin with standard breast and bowel project called trastuzumab herceptin ; to block the chemotherapy developed heart failure. the pittsburgh, pa effects of the her-2 protein. when nsabp published detailed information on herceptin is administered, it interferes with the cardiac toxicity in b-312 and reported "when herceptin is the functions that make a cancer more that an additional 14% of patients did not administered, it aggressive and harder to treat. in 1998 complete the full planned year of herceptin herceptin was approved by the fda for use because of milder weakening of the heart interferes with the in the treatment of metastatic, her-2 muscle. fortunately, it appears that the functions that make positive breast cancer after studies showed heart muscle recovers much of this that when it was combined with weakened function over time. ; it is a cancer more chemotherapy, results were better than with important that patients and physicians aggressive and chemotherapy alone. understand this potential risk; continuing harder to treat." researchers were anxious to determine if follow-up of the women participating in these adding herceptin to standard adjuvant research studies will be important to better therapy would improve outcomes for women with early stage understand the long-term benefits and risk of herceptin. her-2 positive breast cancer. in the spring of 2000, 2 the implications of this research for women diagnosed large studies comparing herceptin + standard with aggressive her-2-positive tumors are substantial. in chemotherapy to standard chemotherapy alone were begun spite of the early nature of these results, the reduction in in the u.s. protocol b-31, conducted by the national risk of recurrence seen with the addition of herceptin to surgical adjuvant breast and bowel project nsabp ; , chemotherapy offers women with such aggressive cancers protocol n9831, coordinated by the north central cancer more effective treatment. women with her-2 positive treatment group ncctg ; , the herceptin adjuvant study breast cancer who are considered candidates for adjuvant hera ; , and breast cancer international study group chemotherapy should now also be considered candidates bcirg ; protocol 006 all looked at various aspects of this for adjuvant herceptin. question. because the b-31 and n9831 studies were quite 1romond eh, perez ea, bryant j, et al trastuzumab plus adjuvant similar, after agreement among the various government chemotherapy for operable her2-positive breast cancer. new engl agencies concerned with the trials, their results were j med 2005; 353: 1673-1684. analyzed together. 2tan-chiu e, yothers g, romond e, et al. assessment of cardiac the results of the b-31 n9831 combined analysis and dysfunction in a randomized trial comparing doxorubicin and the hera trial were striking. both studies showed that cyclophosphamide followed by paclitaxel, with or without trastuzumab as adjuvant therapy in node-positive, human epidermal the addition of herceptin to chemotherapy following growth factor receptor 2-overexpressing breast cancer: nsabp bmastectomy or lumpectomy with radiation reduced the risk 31. j clin oncol 2005; 23: 7811-7819. of death or cancer recurrence in women with her-2 positive dr. geyer thanks barbara c. good, phd, director of scientific breast cancer by roughly 50%. in the b-31 n9831 study, publications for the nsabp, for assistance in the preparation of this 75.4% of women who underwent chemotherapy alone were article. alive and free of disease 3 years after diagnosis; in those.
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